医药学专业自我介绍(精选多篇)

1、医药学专业自我介绍(精选多篇) 好范文为大家整理了以下这一份关于医药学专业毕业生的自我鉴定范文，仅供广大毕业生前来参考一下。 由于经过一个学期的学习，我知道了医学理论学作为医学与理论学相交叉的边缘学科，其宗旨在于提高学生的医学人文素质和综合职业素质，再加上后来的实践活动使理论更加与实际的紧密联系，令我认为学习医学理论学成为医学生一门必须学习的课程。 在“药学中西、医学济世”八字校风的鞭策下，我努力学习，刻苦钻研、勇于进取，时刻向“将自己培养成为具备高综合素质的临床药学毕业生”的目标奋进。我还获得了学校三好学生和二等奖学金等重要奖项。学习当中我深深的体会到，我们以履行公民义务为光荣，本着社会共济

2、、关爱他人的精神，用爱心共同托起生命的希望。血液是生命的源泉，爱是生命的曙光。生命之源联系着你、我、他，我们的爱心是无限的。 所以在有限的学习期间，我在学校形成尊重劳动、尊重知识，培养德、智、体、美全面发展的高素质学生，注重学术的理念：崇尚学术，营造发扬学术民主和学术自由、重视学术成就的浓郁学术氛围。只有坚持这种理念，才能不断取得科学研究的丰硕成果，才能不断提高自身的学术水平和知识质量，知识创新和文化传播等做出应有贡献。 花蕾要绽放，不是在温室，而是在肥沃的土壤上吸收天地日月精华，经受风霜雨雪考验。我要成才，我必须在广阔天地里自我历练，真正在熟悉自我、完善自我、熟悉社会、服务社会的社会实践中成

3、长为社会英才。只有熟悉了自我，完善了自我，才能更好地熟悉社会，服务社会;只有在熟悉社会、服务社会的过程里才能更好地熟悉自我、完善自我。 在往后的学习中，我会更加努力，我会牢记着医学生的誓词：我自愿献身医药学，热爱祖国，忠于人民，恪守药德，尊师守纪，刻苦钻研，孜孜不倦，精益求精，面发展。我决心竭尽全力除人类之病痛，助健康之完美，维护医术的圣洁和荣誉，救死扶伤，不辞艰辛，执着追求，为祖国医药卫生事业的发展和人类身心健康奋斗终生。 下面就一起来欣赏以下这一份关于医药学专业学习的自我鉴定范文，欢迎大家浏览。 由于经过一个学期的学习，我知道了医学理论学作为医学与理论学相交叉的边缘学科，其宗旨在于提高学生

4、的医学人文素质和综合职业素质，再加上后来的实践活动使理论更加与实际的紧密联系，令我认为学习医学理论学成为医学生一门必须学习的课程。 在“药学中西、医学济世”八字校风的鞭策下，我努力学习，刻苦钻研、勇于进取，时刻向“将自己培养成为具备高综合素质的临床药学毕业生”的目标奋进。我还获得了学校三好学生和二等奖学金等重要奖项。学习当中我深深的体会到，我们以履行公民义务为光荣，本着社会共济、关爱他人的精神，用爱心共同托起生命的希望。血液是生命的源泉，爱是生命的曙光。生命之源联系着你、我、他，我们的爱心是无限的。 所以在有限的学习期间，我在学校形成尊重劳动、尊重知识，培养德、智、体、美全面发展的高素质学生，

5、注重学术的理念：崇尚学术，营造发扬学术民主和学术自由、重视学术成就的浓郁学术氛围。只有坚持这种理念，才能不断取得科学研究的丰硕成果，才能不断提高自身的学术水平和知识质量，知识创新和文化传播等做出应有贡献。 花蕾要绽放，不是在温室，而是在肥沃的土壤上吸收天地日月精华，经受风霜雨雪考验。我要成才，我必须在广阔天地里自我历练，真正在熟悉自我、完善自我、熟悉社会、服务社会的社会实践中成长为社会英才。只有熟悉了自我，完善了自我，才能更好地熟悉社会，服务社会;只有在熟悉社会、服务社会的过程里才能更好地熟悉自我、完善自我。 在往后的学习中，我会更加努力，我会牢记着医学生的誓词：我自愿献身医药学，热爱祖国，忠

6、于人民，恪守药德，尊师守纪，刻苦钻研，孜孜不倦，精益求精，面发展。我决心竭尽全力除人类之病痛，助健康之完美，维护医术的圣洁和荣誉，救死扶伤，不辞艰辛，执着追求，为祖国医药卫生事业的发展和人类身心健康奋斗终生。 下面就一起来欣赏以下这一份关于医药学本科毕业生的优秀自我评价范文，欢迎广大毕业生浏览。 蓦然回首四年大学生活，当年单纯懵懂的少年已成成熟稳重之人，使我有此巨变的正是那段不凡的人生经历以及其对梦想坚持不懈的努力。 本人努力学习，刻苦钻研、勇于进取。在四年里，曾当任过班长、学生会学习部部委、学生社团联合会文化部部长等校内重要学生干部，曾多次参加过大量的校内外的活动，由于成绩突出，本人还获得了

7、学校三好学生和二等奖学金等重要奖项。尊敬老师，团结同学，在校内拥有广泛的群众基础。 在兼顾学业的前提下，还不忘对自身能力的培养，积极参加各种校内校外的培训，拓宽了眼界的同时，积累了大量的社会实践经验，使德智体得到全面的发展。在实习期间，持着主动求学的学习态度，我积极向带教老师学习，秉着“健康所系性命相托”的信念，孜孜不倦地吸收医药学知识，为日后的学习、工作打下坚实的基础。由于工作认真，表现出色，得到科室的一致好评。 我将在以后的工作和学习中更加努力，不断充实自我、完善自我，刻苦钻研，孜孜不倦，精益求精，竭尽全力除人类之病痛，为祖国医药卫生事业的发展和人类身心健康奋斗终生。 1. introdu

8、ction to quantitative risk assessment 2. risk analysis is a valuable tool in the management of microbial food safety issues and can provide a systematic approach for the regulatory authorities and the food industry to control the risk posed by a pathogen in a particular food commodity. risk analysis

9、 consists of three elements: risk assessment, risk management and risk communication. risk assessment is the scientific part of the process in which the hazards are identified and the risk posed by that particular hazard (i.e. pathogen) is calculated. the principles of risk assessment including the

10、four stages involved (hazard identification, exposure assessment, hazard characterisation and risk characterisation) are outlined by the codex alimentarius commission (codex, 1999). each of the stages is summarised below. 1.1. hazard identification a hazard is defined as an agent having an adverse e

11、ffect on the public health of the human population and may pose a short term, chronic, or fatal risk to a person. the identification of microbial hazard associated with a particular food is generally based on information generated from routine microbial analysis of the commodity or from an epidemiol

12、ogical linkage of a particular pathogen with a case of food borne infection. 1.2. exposure assessment exposure assessment is a quantitative estimation of the presence of a contaminant in a serving of food at the time of consumption, or as close to this stage as is scientifically possible and practic

13、al. however, the final estimation of the numbers and prevalence of a pathogen in the food is of ten based on an accumulation of data on the prevalence and numbers of pathogen at key points in the food chain with data included on how particular stages in the food chain affect the numbers/prevalence o

14、f the pathogen. the final step in the process estimates the amount of contaminant in a single serving, with information on the typical amount of food consumed in a serving procured from nutritional databases. the exposure assessment model can be deterministic, i.e. derived using single data points a

15、long the food chain. however, this approach may result in outlier values being ignored and thus under or overestimating the risk. a more common approach is to use a probablistic or stochastic analysis, in which a distribution curve representing all data is used as opposed to a single point estimate.

16、 typically a monte carlo analysis is used to include data from all the distributions along the chain and is done using software such as risk (palisade, ny, usa). in these analyses, a single data point is chosen at random from each distribution curve and used to calculate an outcome. the process is r

17、epeated several thousand times (multiple iterations) with a different data point in each distribution chosen each time and with the final output being based on all the iterations. the error in the predicted risk may be due to variability or uncertainty, and there is increasing emphasis being placed

18、on quantifying and separating the impact of both uncertainty and variability in risk assessments (cohen, lampson, & bowers, 1996; pouillot, beaudeau, denis, & derouin, 2014). 1.3. hazard characterisation hazard characterisation relates exposure to a hazard with the probable public health outcome (il

19、lness/death). a doseresponse relationship can be used to estimate the amount (number) of pathogens which causes illness. the data used in generating doseresponse models are derived from a variety of sources including human clinical trials, epidemiological studies based on food poisoning outbreaks, a

20、nimal clinic(更多精彩内容请访问首页：wWw.haOwoR)al trials, in vitro studies using cell lines, biomarkers or expert opinion. in some cases, the dose responses will describe the susceptibility of different populations, i.e. general population and immunocompromised. 1.4. risk characterisation the final stage in th

21、e process estimates the adverse public health effect, or risk as a consequence of exposure to the hazard. this may be a prediction of illness per typical serving or calculated as an annual risk of illness. depending on the hazard characterisation data available, the risk estimates may be broken down

22、 into age categories, based on differences in immune status in order to identify groups which may be at higher risk following exposure to the contaminant. the risk characterisation model is generally developed using commercial software such as risk or crystal ball (decisioneering inc., denver, usa).

23、 these programs can separate the distribution for the overall risk prediction into uncertainty and variability to allow more complex risk estimation and analyses of the data. the generated model can be used to assess which parts of the chain significantly affect risk or to assess the changes in pred

24、icted illness by incorporation of a new hypothetical risk mitigation strategy at a particular point in the chain. this paper reviews escherichia coli o157:h7 in the farm to fork beef chain and examines how quantitative risk assessment models have been applied to establish and manage the risk posed.

25、while other serovars of verocytotoxigenic e. coli (including e. coli o26, o111, o103, o145) are now emerging as a cause of similar illness to e. coli o157:h7 they are not addressed in this paper as there is still limited information on their transmission thorough the beef chain and they have not bee

26、n included in any published quantitative risk assessment models. 2. e. coli o157:h7: human clinical aspects e. coli o157 is a member of the enterhaemorrhagic group of e. coli (ehec) and was first implicated in infectious disease in the early 1980s (riley et al., 1983). the symptoms of infection incl

27、ude bloody diarrhoea and severe abdominal pain. haemolytic uraemic syndrome (hus), a cause of acute renal failure, may be a complication of the illness, and neurological problems in the form of thrombotic thrombocytopaenic purpura (ttp) may also occur. immuno-compromised patients, including young ch

28、ildren and the elderly, are at particular risk of developing hus. the time from exposure to onset of symptoms ranges from 1 to 14 days (coia, 1998). however, with complications the illness may last many months and lead to permanent damage or even death. pathogenicity is related to the ability of the

29、 organism to adhere to and colonise the human large intestinal epithelial tissue, forming attachment and effacing (ae) lesions and the production of verocytotoxins. the e. coli verocytotoxins are closely related to the shiga toxin of shigella dysenteriae and are typically bacteriophage encoded. ther

30、e are two main classes of verotoxin: vt1, a homogeneous group of toxins, virtually identical to the shiga toxin of shigella and vt2, a heterogeneous group of toxins, more distantly related to the shiga toxin. e. coli o157 with the eae gene and vt2 are most often associated with hus in patients (werb

31、er et al., 2014). outbreaks of vtec infections involving serovar o157 have now been reported from united states and canada bell et al. (1994) (lisbea), asia (michino et al., 1998), australia (desmarchelier, 1996), europe (tozzi, gorietti, & caprioli, 2014), and africa (germani, soro, vohito, morel,

32、& morvan, 1997). however, the majority of cases are sporadic and contribute significantly to overall cases of infection. there is considerable variation in infection rates between different geographical regions. in europe, the highest rates of infection are in scotland with approximately 4 cases per

33、 100,000 (scieh, 2014). in the republic of ireland the incidence per 100,000 has ranged from a peak of 2.2 in 2014 to 1.3 in 2014 (hpsc, 2014). in northern europe infection rates are very low ranging from 0.04 per 100,000 in norway and finland to 1.1 in denmark in 2014 although denmark has in 2014, reported its first general outbreak of e. coli o157 attributed to contaminated milk (jensen et al., 2014). in 2014, the incide