医、技学院(华盛顿医疗手册培训-心律失常)课件_第1页
医、技学院(华盛顿医疗手册培训-心律失常)课件_第2页
医、技学院(华盛顿医疗手册培训-心律失常)课件_第3页
医、技学院(华盛顿医疗手册培训-心律失常)课件_第4页
医、技学院(华盛顿医疗手册培训-心律失常)课件_第5页
已阅读5页,还剩103页未读 继续免费阅读

下载本文档

版权说明:本文档由用户提供并上传,收益归属内容提供方,若内容存在侵权,请进行举报或认领

文档简介

CardiacArrhythmiasJunJiangDepartmentofCardiologyCardiacArrhythmiasJunJiang1CardiacArrhythmiasJunJiangCa医、技学院(华盛顿医疗手册培训-心律失常)课件2医、技学院(华盛顿医疗手册培训-心律失常)课件2医、技学院(华盛顿医疗手册培训-心律失常)课件3医、技学院(华盛顿医疗手册培训-心律失常)课件3MechanismsofArrhythmogenesisMechanismsofArrhythmogenesis4MechanismsofArrhythmogenesisTACHYARRHYTHMIASDefinitionCardiacrhythmswhoseventricularrateexceeds100beatsperminute(bpm).ClassificationNarrow-ComplexTachyarrhythmia(QRS<120milliseconds):Wide-ComplexTachyarrhythmia(QRS≥120milliseconds):TACHYARRHYTHMIASDefinition5TACHYARRHYTHMIASDefinitionTACAtrialRe-entryatrialtachycardiaatrialfibrillationatrialflutterAtrio-VentricularRe-entryWolfParkinsonWhitesupraventriculartachycardiaVentricularRe-entryventriculartachycardiaAtrio-VentricularNodalRe-entrysupraventriculartachycardiaRe-entryCircuitsasEctopicFociandArrhythmiaGeneratorsAtrialRe-entryAtrio-Ventricul6AtrialRe-entryAtrio-VentriculHistoryPalpitations(suddenonsetortermination)Dyspnea,angina,lightheadednessorsyncopeanddecreasedlevelofconsciousnessBaselinesymptomsthatreflectpoorLVfunctiondyspneaonexertionOrthopneaparoxysmalnocturnaldyspnealowerextremityswellingHistoryoforganicheartdiseaseorendocrinopathyHistoryoffamilialorcongenitalcausesofarrhythmiashypertrophiccardiomyopathy(HCM)congenitallongQTsyndromeMedications:Criticaltoobtainacompletelist,includingover-the-counterandherbalmedicationsHistory7HistoryHistory7PhysicalExaminationSignsofclinicalstabilityorinstabilityvitalsignsmentalstatusperipheralperfusionFindingsoforganicheartdiseaseJVPpulmonaryralesperipheraledemaCardiacborderS3gallopmurmurPalpatethepulseandassessforrateandregularity.“Cannon”AwavesIfirregular,thensuggestiveofunderlyingAVdissociationandclueforVTIfregularin1:1ratiowithperipheralpulse,thensuggestiveofAVNRT,AVRT,orajunctionaltachycardiaPhysicalExamination8PhysicalExaminationPhysicalEDiagnosticTestingLaboratoriesSerumelectrolytescompletebloodcount(CBC)thyroidfunctiontestsatoxicologyscreenElectrocardiographyImagingChestradiographEchocardiogramCT,MRI,CAGContinuousambulatoryECGmonitoringIn-hospitaltelemetrymonitoringEventrecordersExerciseECGElectrophysiologystudy(EPS)DiagnosticTesting9DiagnosticTestingDiagnosticTTreatmentTreatment10TreatmentTreatment10

11

RhythmsfromtheSinusNodeNormalSinusRhythm(NSR)

SinusTachycardia:HR>100b/mCauses:Withdrawalofvagultone&Sympatheticstimulation(exercise,pain,orfight)Fever&inflammationHypovolemiaAnemaiHypoxiaHeartFailureorCardiogenicShock(bothrepresenthypoperfusionstates)HeartAttack(myocardialinfarctionorextensionofinfarction)Drugs(alcohol,nicotine,caffeine)TherapytargetedattreatmentofunderlyingpathophysiologicprocessRhythmsfromtheSinusNodeNor12RhythmsfromtheSinusNodeNorSupraventricularTachyarrhythmiasParoxysmalsupraventriculartachycardi(PSVT)PrevalenceandincidenceofPSVTare2.25per1,000AVNRT(60%)AVRT(30%)AtrialfibrillationAFisthemostcommonnarrow-complextachycardiaseenintheinpatientsettingAtrialflutterAFlcanoftenaccompanyAFandisdiagnosedone-tenthasoftenasAFbutistwiceasprevalentasthePSVTsAtrialtachycardiafarlesscommonJunctionaltachycardiaSinoatrialnodalreentranttachycardia(SANRT)SupraventricularTachyarrhythm13SupraventricularTachyarrhythmTREATMENTAcutetreatmentofsymptomaticSVTshouldfollowtheACLSprotocolasbeforeAVnodalblockingagentsortechniquesManySVTscanbeterminatedAF,AFl,andsomeatrialtachycardiaswillpersistwithaslowingoftheventricularrateCorrectionofelectrolyteabnormalities(K+andMg+)UnderlyingetiologyChronictreatmentshouldbeaimedateitherpreventionofrecurrenceorpreventionofthecomplicationsRadiofrequencyablation(RFA)Successratesfrom85%to95%Comparedtoantiarrhythmictherapy,RFAimprovesqualityoflifeandismorecost-effectiveinthelongtermTREATMENT14TREATMENTTREATMENT1415AVNRTP’inleadI,II,V1-V315AVNRTP’inleadI,II,V1-V31515AVNRTP’inleadI,II,V1-V31AVRTAVRT16AVRTAVRT16WPW-A4WPW-A417WPW-A4WPW-A417WPW-BWPW-B18WPW-BWPW-B18AtrialFibrillationClassificationFirstoccurrence.Thespontaneousconversionrateis>60%ParoxysmalAF:<7daysandusually<48hoursinduration.PersistentAF:>7daysindurationorrequirecardioversionPermanentAF

MedicalmanagementRatecontrolofAFdiltiazem,verapamilβ-adrenergicblockersdigoxinPreventionofthromboembolicevents

RhythmcontrolPharmacologiccontrolElectricalcardioversionNonpharmacologicmethodsofrhythmcontrolincludecatheterorsurgicalablationAtrialFibrillation19AtrialFibrillationAtrialFibrClassificationofAnti-arrhythmicsClassificationofAnti-arrhyth20ClassificationofAnti-arrhyth医、技学院(华盛顿医疗手册培训-心律失常)课件21医、技学院(华盛顿医疗手册培训-心律失常)课件21StrokeRiskinPatientsWithNonvalvularAFStrokeRiskinPatientsWithN22StrokeRiskinPatientsWithN23AFwithWPW23AFwithWPW2323AFwithWPW23AFwithWPW23医、技学院(华盛顿医疗手册培训-心律失常)课件24医、技学院(华盛顿医疗手册培训-心律失常)课件24医、技学院(华盛顿医疗手册培训-心律失常)课件25医、技学院(华盛顿医疗手册培训-心律失常)课件25thereisnopwave,indicatingthatitdidnotoriginateanywhereintheatria,butsincetheQRScomplexisstillthinandnormallooking,wecanconcludethatthebeatoriginatedsomewhereneartheAVjunction.Thebeatisthereforecalleda"junctional"ora“nodal”beatJunctionalEscapeBeatQRSisslightlydifferentbutstillnarrow,indicatingthatconductionthroughtheventricleisrelativelynormalRecognizingandNamingBeats&Rhythmsthereisnopwave,indicating26thereisnopwave,indicatingVentricularTachyarrhythmiasGENERALPRINCIPLESVentriculartachyarrhythmiasshouldbeinitiallyapproachedwiththeassumptionthattheywillhaveamalignantcourseuntilprovenotherwiseCharacterizationofthearrhythmiainvolveshemodynamicstabilityDurationMorphologythepresenceorlackofunderlyingstructuralheartdiseaseUltimately,thischaracterizationwillaidindeterminingthepatient'sriskforsuddencardiacarrestandneedfordeviceorablation-basedtherapyVentricularTachyarrhythmias27VentricularTachyarrhythmiasVeDefinitionofVentricularTachyarrhythmiasNonsustainedVTThreeormoreconsecutiveventricularcomplexes(>100bpm)thatterminatesspontaneouslywithin30secondswithoutsignificanthemodynamicconsequencesorneedforinterventionSustainedmonomorphicVTTachycardiacomposedofventricularcomplexesofasingleQRSmorphologythatlastslongerthan30secondsorrequirescardioversionduetohemodynamiccompromise.PolymorphicVTischaracterizedbyanever-changingQRSmorphologyTdPistypicallyprecededbyaprolongedQTintervalinsinusrhythmPolymorphicVTisusuallyassociatedwithhemodynamiccollapseorinstabilityVFisassociatedwithdisorganizedmechanicalcontraction,hemodynamiccollapse,andsuddendeathSCDisdefinedasthedeaththatoccurswithin1houroftheonsetofsymptomsIntheUnitedStates,350,000casesofSCDoccurannuallyDefinitionofVentricularTach28DefinitionofVentricularTach医、技学院(华盛顿医疗手册培训-心律失常)课件29医、技学院(华盛顿医疗手册培训-心律失常)课件29医、技学院(华盛顿医疗手册培训-心律失常)课件30医、技学院(华盛顿医疗手册培训-心律失常)课件30医、技学院(华盛顿医疗手册培训-心律失常)课件31医、技学院(华盛顿医疗手册培训-心律失常)课件31EtiologyVTassociatedwithstructuralheartdiseaseActiveischemiaorhistoryofinfarctNonischemiccardiomyopathyInfiltrativecardiomyopathies(sarcoid,hemochromatosis,amyloid)AdultswithpriorrepairofcongenitalheartdiseaseArrhythmogenicrightventriculardysplasiaorcardiomyopathyBundlebranchreentryVTVTintheabsenceofstructuralheartdiseaseInheritedionchannelopathies(Brugada,longQTsyndromes)CatecholaminergicpolymorphicVTIdiopathicVT(VOT)Etiology32EtiologyEtiology32BrugadacriteriaBrugadacriteria33BrugadacriteriaBrugadacriteRecognizingandNamingBeats&RhythmsNotesonV-tach:CausesofV-tach

PriorMI,CAD,dilatedcardiomyopathy,oritmaybeidiopathic(noknowncause)TypicalV-tachpatient

MIwithcomplications&extensivenecrosis,EF<40%,dwallmotion,v-aneurysm)V-tachcomplexesarelikelytobesimilarandtherhythmregularIrregularV-Tachrhythmsmaybeduetoto:breakthroughofatrialconductionatriamay“capture”theentirebeatbeatanatrialbeatmay“merge”withanectopicventricularbeat(fusionbeat)Fusionbeat-notep-waveinfrontofPVCandthePVCisnarrowerthantheotherPVC’s–thisindicatesthebeatisaproductofboththesinusnodeandanectopicventricularfocusCapturebeat-notethatthecomplexisnarrowenoughtosuggestnormalventricularconduction.Thisindicatesthatanatrialimpulsehasmadeitthroughandconductionthroughtheventriclesisrelativelynormal.RecognizingandNamingBeats&34RecognizingandNamingBeats&医、技学院(华盛顿医疗手册培训-心律失常)课件35医、技学院(华盛顿医疗手册培训-心律失常)课件35医、技学院(华盛顿医疗手册培训-心律失常)课件36医、技学院(华盛顿医疗手册培训-心律失常)课件36TREATMENTDifferentiationofSVTwithaberrancyfromVTonthebasisofanalysisofthesurfaceECGiscriticalinthedeterminationofappropriateacuteandchronictherapyImmediateunsynchronizedDCcardioversionistheprimarytherapyforpulselessVTandVFNonpharmacologictherapyICDsRadiofrequencycatheterablationMedicationsVFthatisresistanttoexternaldefibrillationrequirestheadditionofIVantiarrhythmicagents.IVamiodaroneappearstobemoreeffectiveinincreasingsurvivalofVFwhenusedinconjunctionwithdefibrillationChronicantiarrhythmicdrugtherapyisindicatedforthetreatmentofrecurrentsymptomaticventriculararrhythmiasTREATMENT37TREATMENTTREATMENT37医、技学院(华盛顿医疗手册培训-心律失常)课件38医、技学院(华盛顿医疗手册培训-心律失常)课件38LAORAOLAORAO39LAORAOLAORAO39BRADYARRHYTHMIASDefinitionCardiacrhythmswhoseventricularrate<60bpmCausesofBradycardiaIntrinsicCongenitaldiseaseIdiopathicdegeneration(aging)InfarctionorischemiaCardiomyopathyInfiltrativedisease:sarcoidosis,amyloidosisCollagenvasculardiseasesSurgicaltraumaInfectiousdiseaseExtrinsicAutonomicallymediated(NeurocardiogenicsyncopeCarotidsinushypersensitivity)Increasedvagaltone:coughing,vomiting,micturition,defecation,intubationDrugs:β-blockers,calciumchannelblockers,digoxin,antiarrhythmicagentsHypothyroidismHypothermiaNeurologicdisorders:increasedintracranialpressureElectrolyteimbalances:hyperkalemia,hypermagnesemiaHypercarbia/obstructivesleepapneaSepsisBRADYARRHYTHMIASDefinition40BRADYARRHYTHMIASDefinitionBRADIAGNOSISSTABLE:Isthepatienthemodynamicallyunstable?SYMPTOMS:Doesthepatienthavesymptomsanddothesymptomscorrelatewiththebradycardia?SHORT-TERM:Arethecircumstancessurroundingthearrhythmiareversibleortransient?SOURCE:Whereintheconductionsystemisthedysfunction?Hasthebradyarrhythmiabeencapturedonelectrocardiographicmonitoring?SCHEDULEAPACEMAKER:DoesthepatientrequireaPPM?DIAGNOSIS41DIAGNOSISDIAGNOSIS41

SinusBradycardia:HR<60b/mCauses:Increasedvagultone,decreasedsympatheticoutput,(endurancetraining)HypothyroidismHeartAttack(commonininferiorwallinfarction)Vasovagulsyncope(peoplepassingoutwhentheygettheirblooddrawn)Depression

SickSinusSyndrome:Failureoftheheart’spacemakingcapabilitiesCauses:Idiopathic(nocausecanbefound)Cardiomyopathy(diseaseandmalformationofthecardiacmuscle)ImplicationsandAssociationsAssociatedwithTachycardia/BradycardiaarrhythmiasIsoftenfollowedbyanectopic“escapebeat”oranectopic“rhythm”SinusBradycardia:HR<60b42SinusBradycardia:HR<60b医、技学院(华盛顿医疗手册培训-心律失常)课件43医、技学院(华盛顿医疗手册培训-心律失常)课件43医、技学院(华盛顿医疗手册培训-心律失常)课件44医、技学院(华盛顿医疗手册培训-心律失常)课件44医、技学院(华盛顿医疗手册培训-心律失常)课件45医、技学院(华盛顿医疗手册培训-心律失常)课件45医、技学院(华盛顿医疗手册培训-心律失常)课件46医、技学院(华盛顿医疗手册培训-心律失常)课件46医、技学院(华盛顿医疗手册培训-心律失常)课件47医、技学院(华盛顿医疗手册培训-心律失常)课件47医、技学院(华盛顿医疗手册培训-心律失常)课件48医、技学院(华盛顿医疗手册培训-心律失常)课件48医、技学院(华盛顿医疗手册培训-心律失常)课件49医、技学院(华盛顿医疗手册培训-心律失常)课件49医、技学院(华盛顿医疗手册培训-心律失常)课件50医、技学院(华盛顿医疗手册培训-心律失常)课件50医、技学院(华盛顿医疗手册培训-心律失常)课件51医、技学院(华盛顿医疗手册培训-心律失常)课件51医、技学院(华盛顿医疗手册培训-心律失常)课件52医、技学院(华盛顿医疗手册培训-心律失常)课件52actuallya"retrogradep-wavemaysometimesbeseenontherighthandsideofbeatsthatoriginateintheventricles,indicatingthatdepolarizationhasspreadbackupthroughtheatriafromtheventriclesQRSiswideandmuchdifferent("bizarre")lookingthanthenormalbeats.Thisindicatesthatthebeatoriginatedsomewhereintheventriclesandconsequently,conductionthroughtheventriclesdidnottakeplacethroughnormalpathways.Itisthereforecalleda“ventricular”beatVentricularEscapeBeatthereisnopwave,indicatingthatthebeatdidnotoriginateanywhereintheatriaRecognizingandNamingBeats&Rhythmsactuallya"retrogradep-wave53actuallya"retrogradep-waveTHANKSTHANKS54THANKSTHANKS54CardiacArrhythmiasJunJiangDepartmentofCardiologyCardiacArrhythmiasJunJiang55CardiacArrhythmiasJunJiangCa医、技学院(华盛顿医疗手册培训-心律失常)课件56医、技学院(华盛顿医疗手册培训-心律失常)课件56医、技学院(华盛顿医疗手册培训-心律失常)课件57医、技学院(华盛顿医疗手册培训-心律失常)课件57MechanismsofArrhythmogenesisMechanismsofArrhythmogenesis58MechanismsofArrhythmogenesisTACHYARRHYTHMIASDefinitionCardiacrhythmswhoseventricularrateexceeds100beatsperminute(bpm).ClassificationNarrow-ComplexTachyarrhythmia(QRS<120milliseconds):Wide-ComplexTachyarrhythmia(QRS≥120milliseconds):TACHYARRHYTHMIASDefinition59TACHYARRHYTHMIASDefinitionTACAtrialRe-entryatrialtachycardiaatrialfibrillationatrialflutterAtrio-VentricularRe-entryWolfParkinsonWhitesupraventriculartachycardiaVentricularRe-entryventriculartachycardiaAtrio-VentricularNodalRe-entrysupraventriculartachycardiaRe-entryCircuitsasEctopicFociandArrhythmiaGeneratorsAtrialRe-entryAtrio-Ventricul60AtrialRe-entryAtrio-VentriculHistoryPalpitations(suddenonsetortermination)Dyspnea,angina,lightheadednessorsyncopeanddecreasedlevelofconsciousnessBaselinesymptomsthatreflectpoorLVfunctiondyspneaonexertionOrthopneaparoxysmalnocturnaldyspnealowerextremityswellingHistoryoforganicheartdiseaseorendocrinopathyHistoryoffamilialorcongenitalcausesofarrhythmiashypertrophiccardiomyopathy(HCM)congenitallongQTsyndromeMedications:Criticaltoobtainacompletelist,includingover-the-counterandherbalmedicationsHistory61HistoryHistory61PhysicalExaminationSignsofclinicalstabilityorinstabilityvitalsignsmentalstatusperipheralperfusionFindingsoforganicheartdiseaseJVPpulmonaryralesperipheraledemaCardiacborderS3gallopmurmurPalpatethepulseandassessforrateandregularity.“Cannon”AwavesIfirregular,thensuggestiveofunderlyingAVdissociationandclueforVTIfregularin1:1ratiowithperipheralpulse,thensuggestiveofAVNRT,AVRT,orajunctionaltachycardiaPhysicalExamination62PhysicalExaminationPhysicalEDiagnosticTestingLaboratoriesSerumelectrolytescompletebloodcount(CBC)thyroidfunctiontestsatoxicologyscreenElectrocardiographyImagingChestradiographEchocardiogramCT,MRI,CAGContinuousambulatoryECGmonitoringIn-hospitaltelemetrymonitoringEventrecordersExerciseECGElectrophysiologystudy(EPS)DiagnosticTesting63DiagnosticTestingDiagnosticTTreatmentTreatment64TreatmentTreatment64

65

RhythmsfromtheSinusNodeNormalSinusRhythm(NSR)

SinusTachycardia:HR>100b/mCauses:Withdrawalofvagultone&Sympatheticstimulation(exercise,pain,orfight)Fever&inflammationHypovolemiaAnemaiHypoxiaHeartFailureorCardiogenicShock(bothrepresenthypoperfusionstates)HeartAttack(myocardialinfarctionorextensionofinfarction)Drugs(alcohol,nicotine,caffeine)TherapytargetedattreatmentofunderlyingpathophysiologicprocessRhythmsfromtheSinusNodeNor66RhythmsfromtheSinusNodeNorSupraventricularTachyarrhythmiasParoxysmalsupraventriculartachycardi(PSVT)PrevalenceandincidenceofPSVTare2.25per1,000AVNRT(60%)AVRT(30%)AtrialfibrillationAFisthemostcommonnarrow-complextachycardiaseenintheinpatientsettingAtrialflutterAFlcanoftenaccompanyAFandisdiagnosedone-tenthasoftenasAFbutistwiceasprevalentasthePSVTsAtrialtachycardiafarlesscommonJunctionaltachycardiaSinoatrialnodalreentranttachycardia(SANRT)SupraventricularTachyarrhythm67SupraventricularTachyarrhythmTREATMENTAcutetreatmentofsymptomaticSVTshouldfollowtheACLSprotocolasbeforeAVnodalblockingagentsortechniquesManySVTscanbeterminatedAF,AFl,andsomeatrialtachycardiaswillpersistwithaslowingoftheventricularrateCorrectionofelectrolyteabnormalities(K+andMg+)UnderlyingetiologyChronictreatmentshouldbeaimedateitherpreventionofrecurrenceorpreventionofthecomplicationsRadiofrequencyablation(RFA)Successratesfrom85%to95%Comparedtoantiarrhythmictherapy,RFAimprovesqualityoflifeandismorecost-effectiveinthelongtermTREATMENT68TREATMENTTREATMENT6869AVNRTP’inleadI,II,V1-V315AVNRTP’inleadI,II,V1-V36969AVNRTP’inleadI,II,V1-V31AVRTAVRT70AVRTAVRT70WPW-A4WPW-A471WPW-A4WPW-A471WPW-BWPW-B72WPW-BWPW-B72AtrialFibrillationClassificationFirstoccurrence.Thespontaneousconversionrateis>60%ParoxysmalAF:<7daysandusually<48hoursinduration.PersistentAF:>7daysindurationorrequirecardioversionPermanentAF

MedicalmanagementRatecontrolofAFdiltiazem,verapamilβ-adrenergicblockersdigoxinPreventionofthromboembolicevents

RhythmcontrolPharmacologiccontrolElectricalcardioversionNonpharmacologicmethodsofrhythmcontrolincludecatheterorsurgicalablationAtrialFibrillation73AtrialFibrillationAtrialFibrClassificationofAnti-arrhythmicsClassificationofAnti-arrhyth74ClassificationofAnti-arrhyth医、技学院(华盛顿医疗手册培训-心律失常)课件75医、技学院(华盛顿医疗手册培训-心律失常)课件75StrokeRiskinPatientsWithNonvalvularAFStrokeRiskinPatientsWithN76StrokeRiskinPatientsWithN77AFwithWPW23AFwithWPW7777AFwithWPW23AFwithWPW77医、技学院(华盛顿医疗手册培训-心律失常)课件78医、技学院(华盛顿医疗手册培训-心律失常)课件78医、技学院(华盛顿医疗手册培训-心律失常)课件79医、技学院(华盛顿医疗手册培训-心律失常)课件79thereisnopwave,indicatingthatitdidnotoriginateanywhereintheatria,butsincetheQRScomplexisstillthinandnormallooking,wecanconcludethatthebeatoriginatedsomewhereneartheAVjunction.Thebeatisthereforecalleda"junctional"ora“nodal”beatJunctionalEscapeBeatQRSisslightlydifferentbutstillnarrow,indicatingthatconductionthroughtheventricleisrelativelynormalRecognizingandNamingBeats&Rhythmsthereisnopwave,indicating80thereisnopwave,indicatingVentricularTachyarrhythmiasGENERALPRINCIPLESVentriculartachyarrhythmiasshouldbeinitiallyapproachedwiththeassumptionthattheywillhaveamalignantcourseuntilprovenotherwiseCharacterizationofthearrhythmiainvolveshemodynamicstabilityDurationMorphologythepresenceorlackofunderlyingstructuralheartdiseaseUltimately,thischaracterizationwillaidindeterminingthepatient'sriskforsuddencardiacarrestandneedfordeviceorablation-basedtherapyVentricularTachyarrhythmias81VentricularTachyarrhythmiasVeDefinitionofVentricularTachyarrhythmiasNonsustainedVTThreeormoreconsecutiveventricularcomplexes(>100bpm)thatterminatesspontaneouslywithin30secondswithoutsignificanthemodynamicconsequencesorneedforinterventionSustainedmonomorphicVTTachycardiacomposedofventricularcomplexesofasingleQRSmorphologythatlastslongerthan30secondsorrequirescardioversionduetohemodynamiccompromise.PolymorphicVTischaracterizedbyanever-changingQRSmorphologyTdPistypicallyprecededbyaprolongedQTintervalinsinusrhythmPolymorphicVTisusuallyassociatedwithhemodynamiccollapseorinstabilityVFisassociatedwithdisorganizedmechanicalcontraction,hemodynamiccollapse,andsuddendeathSCDisdefinedasthedeaththatoccurswithin1houroftheonsetofsymptomsIntheUnitedStates,350,000casesofSCDoccurannuallyDefinitionofVentricularTach82DefinitionofVentricularTach医、技学院(华盛顿医疗手册培训-心律失常)课件83医、技学院(华盛顿医疗手册培训-心律失常)课件83医、技学院(华盛顿医疗手册培训-心律失常)课件84医、技学院(华盛顿医疗手册培训-心律失常)课件84医、技学院(华盛顿医疗手册培训-心律失常)课件85医、技学院(华盛顿医疗手册培训-心律失常)课件85EtiologyVTassociatedwithstructuralheartdiseaseActiveischemiaorhistoryofinfarctNonischemiccardiomyopathyInfiltrativecardiomyopathies(sarcoid,hemochromatosis,amyloid)AdultswithpriorrepairofcongenitalheartdiseaseArrhythmogenicrightventriculardysplasiaorcardiomyopathyBundlebranchreentryVTVTintheabsenceofstructuralheartdiseaseInheritedionchannelopathies(Brugada,longQTsyndromes)CatecholaminergicpolymorphicVTIdiopathicVT(VOT)Etiology86EtiologyEtiology86BrugadacriteriaBrugadacriteria87BrugadacriteriaBrugadacriteRecognizingandNamingBeats&RhythmsNotesonV-tach:CausesofV-tach

PriorMI,CAD,dilatedcardiomyopathy,oritmaybeidiopathic(noknowncause)TypicalV-tachpatient

MIwithcomplications&extensivenecrosis,EF<40%,dwallmotion,v-aneurysm)V-tachcomplexesarelikelytobesimilarandtherhythmregularIrregularV-Tachrhythmsmaybeduetoto:breakthroughofatrialconductionatriamay“capture”theentirebeatbeatanatrialbeatmay“merge”withanectopicventricularbeat(fusionbeat)Fusionbeat-notep-waveinfrontofPVCandthePVCisnarrowerthantheotherPVC’s–thisindicatesthebeatisaproductofboththesinusnodeandanectopicventricularfocusCapturebeat-notethatthecomplexisnarrowenoughtosuggestnormalventricularconduction.Thisindicatesthatanatrialimpulsehasmadeitthroughandconductionthroughtheventriclesisrelativelynormal.RecognizingandNamingBeats&88RecognizingandNamingBeats&医、技学院(华盛顿医疗手册培训-心律失常)课件89医、技学院(华盛顿医疗手册培训-心律失常)课件89医、技学院(华盛顿医疗手册培训-心律失常)课件90医、技学院(华盛顿医疗手册培训-心律失常)课件90TREATMENTDifferentiationofSVTwithaberrancyfromVTonthebasisofanalysisofthesurfaceECGiscriticalinthedeterminationofappropriateacuteandchronictherapyImmediateunsynchronizedDCcardioversionistheprimarytherapyforpulselessVTandVFNonpharmacologictherapyICDsRadiofrequencycatheterablationMedicationsVFthatisresistanttoexternaldefibrillationrequirestheadditionofIVantiarrhythmicagents.IVamiodaroneappearstobemoreeffectiveinincreasingsurvivalofVFwhenusedinconjunctionwithdefibrillationChronicantiarrhythmicdrugtherapyisindicatedforthetreatmentofrecurrentsymptomaticventriculararrhythmiasTREATMENT91TREATMENTTREATMENT91医、技学院(华盛顿医疗手册培训-心律失常)课件92医、技学院(华盛顿医疗手册培训-心律失常)课件92LAORAOLAORAO93LAORAOLAORAO93BRADYARRHYTHMIASDefinitionCardiacrhythmswhoseventricularrate<60bpmCausesofBradycardiaIntrinsicCongenitaldiseaseIdiopathicdegeneration(aging)InfarctionorischemiaCardiomyopathyInfiltrativedisease:sarcoidosis,amyloidosisCollagenvasculardiseasesSurgicaltraumaInfectiousdiseaseExtrinsicAutonomicallymediated(NeurocardiogenicsyncopeCarotidsinushypersensitivity)Increasedvagaltone:coughing,vomiting,micturition,defecation,intubationDrugs:β-blockers,calci

温馨提示

  • 1. 本站所有资源如无特殊说明,都需要本地电脑安装OFFICE2007和PDF阅读器。图纸软件为CAD,CAXA,PROE,UG,SolidWorks等.压缩文件请下载最新的WinRAR软件解压。
  • 2. 本站的文档不包含任何第三方提供的附件图纸等,如果需要附件,请联系上传者。文件的所有权益归上传用户所有。
  • 3. 本站RAR压缩包中若带图纸,网页内容里面会有图纸预览,若没有图纸预览就没有图纸。
  • 4. 未经权益所有人同意不得将文件中的内容挪作商业或盈利用途。
  • 5. 人人文库网仅提供信息存储空间,仅对用户上传内容的表现方式做保护处理,对用户上传分享的文档内容本身不做任何修改或编辑,并不能对任何下载内容负责。
  • 6. 下载文件中如有侵权或不适当内容,请与我们联系,我们立即纠正。
  • 7. 本站不保证下载资源的准确性、安全性和完整性, 同时也不承担用户因使用这些下载资源对自己和他人造成任何形式的伤害或损失。

评论

0/150

提交评论